||Yun-Ying Zhao, Chun-Lei Cao, Ying-Li Liu, Jing Wang, Jie Li, Shi-Yun Li, Yu Deng
||2 We showed that the intracellular ROS levels in 51 mutants were increased by high zinc stress, suggesting their possible involvement in regulating ROS homeostasis in response to high zinc. The results also revealed that excess zinc could generate oxidative damage and then activate the expression of several antioxidant defenses genes. Taken together, the data obtained indicated that excess zinc toxicity might be mainly due to the high intracellular zinc levels and ROS levels induced by zinc stress in yeast cells. Our current findings would provide a basis to understand the molecular mechanisms of zinc toxicity in yeast cells.
||G3 (Bethesda) 2020; 10:479-488
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